By A. F. Junod (auth.), Dr. J. L. Vincent (eds.)
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Additional info for 6th International Symposium on Intensive Care and Emergency Medicine: Brussels, Belgium, April 15–18, 1986
Minty BD, Scudder C, Grantham C, Jones JG, Bakhle YS, (1985) Changes in pulmonary permeability and metabolic funtion following ANTU lung injury. Amer Rev Respir Dis 131 :A340 15. Morel D, Dargent F, Bachmann M, Suter P, Junod AF. (1985) Pulmonary extraction of serotonin and propranolol in patients with adult respiratory distress syndrome. Am Rev Respir Dis 132:479-484 17. Weibel ER (1984) The pathway for oxygen. Harvard University Press, Cambridge, Mass and London p 231-271 18. Weinberg PF, Matthay MA, Webster RO, Roskos KV, Goldstein 1M, and Murray JF.
Boysen same time, pulmonary artery pressure increases, as does both the systolic and diastolic pressure. Central venous pressure also increases but there is an overall increase in pulmonary vascular resistance. Cardiac output decreases, but not severely since, with higher volumes of aspiration of freshwater, there is some movement of fluid into the vascular space with the effect of some protection of cardiac output. Mixed venous oxygen saturation during the same time decreases precipitously, again, partly due to gas exchange in the lung and partly due to decreased cardiac output.
Lung lavage. The Gallium scan localises areas of acute or chronic inflammation at the alveolar level and is not influenced by inflammatory airway disease. Gallium - 67 citrate is given intravenously and the patient is scanned 48 hours later. The mechanisms of gallium - 67 uptake in regions of inflammation are unknown but most of the gallium is taken up in macrophages with only a small uptake in neutrophils. Most studies of Gallium - 67 uptake have been reported in patients with sarcoidosis or in fibrosing alveolitis.
6th International Symposium on Intensive Care and Emergency Medicine: Brussels, Belgium, April 15–18, 1986 by A. F. Junod (auth.), Dr. J. L. Vincent (eds.)